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Future Possibilities



Neurons do not die, but could be reactivated!


A research by top university and published in a top journal may potentially change the course of the disease as well as the patient. It concluded that the disease not just affect the dopaminergic neurons (axons), but affect the body of cell itself (soma). Also it observed that the neurons affected by disease do not die, they only lose some of the properties.

Consequently, they could be reactivated with potentially new and future treatments.


Following is the link for original source:

https://www.news-medical.net/news/20211108/Research-opens-the-door-to-new-therapeutic-options-for-Parkinsons-disease.aspx



An integrated approach to treatment may be needed


With improved detection and wearable technologies, there is need to develop individualized and on-demand therapies. While there are several pharmacological approaches (e.g., dopaminergic and beyond dopamine), non-pharmacological approaches (physiotherapy and cueing, cognitive training, and non-invasive brain stimulation) and surgical approaches to freezing (i.e., dual-site deep brain stimulation, closed-loop programming), an integrated collaborative approach will be necessary to investigate new therapeutic avenues.


Following is the link to the source:

https://www.frontiersin.org/articles/10.3389/fnhum.2021.741918/full



Gut microbiota and Parkinson's Disease may be linked

Through research, scientists have got evidence that link gut microbiota ot symptoms and pathophysiology of patients of Parkinson's Disease. A recent study highlighted significant connection between levels of certain types of gut microbiota and Parkinson's Disease.


Following is the link to original work:

https://ejnpn.springeropen.com/articles/10.1186/s41983-021-00407-z



Visual impairment could be an early indicator


A recent study found that people with visual impairment (Habitual distance visual acuity) have an increased risk of incidence of Parkinson's Disease. It concludes that visual impairment may serve as a modifiable risk factor for prevention of future Parkinson's Disease.


Following is the link to the original paper:

https://www.thelancet.com/journals/eclinm/article/PIIS2589-5370(21)00470-3/fulltext



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